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Soma Medication: Carisoprodol Uses, Tests, Side Effects and Treatment


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Carisoprodol (trade name Soma) can be a centrally-acting muscle relaxant. Carisoprodol is slightly soluble in water and is soluble in alcohol, chloroform, and slurry independently. The solubility of the drug is very free of hydrogen ion concentrations. Carisoprodol is manufactured and marketed in the US by the Iraqi National Congress of Medicine prescription drugs. Full names Sanoma and Carisoma under the brand Soma and the UK and various countries. Soma medication is mixed by itself or with analgesics and also in a preparation (soma compound with coda) with iodine and alkaloids.


On June 1, 1959, several Yankee pharmacists convened an urban center at Wayne State University in Michigan to debate a brand new drug. The drug, originally thought to have antiseptic properties, was found to have central muscle relaxant properties. Dr. Frank M. Berger developed at Wallace Laboratories and was named Carisoprodol.

Carisoprodol was developed to support a minor tranquilizer, with the hope that it may have a higher muscle calming properties, lower potential for abuse, and a smaller tranquilizer at lower risk of the drug. The substitution of 1 atom with an isopropyl cluster of associate degree on one between carbamyl nitrogenates was about to produce a molecule with new pharmacologic properties.

The brand Soma is shared with Soma/Hyoma of Ancient India, a medicine described in ancient Indo-Aryan writings. Various classical and fashionable researchers have proven that soma can be anything from cedar or alcoholic combined with mushrooms of the genus Amanita with psychotropic effects. Anti-cholinergic-drug in which the platter is still an unknown psychoactive drug, an extractive ally of the unknown chemical category, lowly or drug or origin, or perhaps coca or different drug from the new world, so far unknown pre-Viking, pre-Columbian contact.

Soma is also the name of the fictional drug portrayed in the brave new world of Aldus Huxley.

Usage and Options

However Scandinavian country reports have shown that carisoprodol has the potential for abuse as a minor tranquilizer and/or potentiator of hydrocodone, dihydrocodeine, codeine, and similar drugs. It is prescribed in North America, to relax muscles and relax muscles. In Europe, doctors favor muscle relaxation. Within the UK, the benzodiazepines area unit is best preferred instead. From all the upper and chlorzoxazone area units employed in Canada.

As of November 2007, carisoprodol (Somadril, Somadril comp.) Has been introduced in Sweden due to market dependence and side effects. The agency overseeing the prescription drug has thought of various drugs, which are used with continued indication, as carisoprodol to have a sustained or high effect when the drug is not at risk. In 2008 it was also launched in the Scandinavian country.

In the EU, the ECU Medicines Agency has issued an unnecessary statement that member countries suspend the promotion of authorization for this product within the treatment of acute (not old) back pain.

In the US, while carisoprodol is not a drug below federal regulations, as of February 2010, carisoprodol is taken as a Schedule IV drug by the states of Alabama, Arizona, Arkansas, Florida, Georgia, Hawaii, Indiana, Kentucky. Louisiana, Massachusetts, Minnesota, Mississippi, New Mexico, Nevada, Oklahoma. The Lone-Star State (prescribing the state's new drug program requires a physician to be embraced and embraced, and includes a state "DPS" range as a Drug Enforcement Agency on all controlled substance prescriptions), Utah and Washington. It is a Schedule III drug in WV. The remainder of the US, except for more than the designated states, falls below the Drug Enforcement Agency programming for the drug, which considers carisoprodol a non-prescriptive chemical, meaning that carisoprodol is a generic prescription by the US centralized Is taken as a medicine, with inspection provided solely by the US Food and Drug Administration (FDA).

On March 26, 2010, the Drug Enforcement Agency issued information about the planned hearing on planned control, in which the Schedule IV of the Controlled Substances Act was told about the position of carisoprodol.

Absorbers of carisoprodol sometimes ask for its possible divisibility, enthusiasm, and significant greed, comfort, and anxious effects. Also, due to its potent effects on narcotics, it is commonly misused in combination with many opioid medications.

With most psychological substances, tolerance will type quickly. This culprit causes a constant increase in the dose to get the necessary effect. Like any medicine, it can be dangerous for a large number of reasons. For this reason, people with an addiction background should not be prescribed.


  • Sedation,
  • Tandoori,
  • Anxiolysis,
  • Lack of soreness,
  • Muscular relaxation (and relief from hypertonia).

Side Effects

A normal dose of 350mg is not likely to have side effects different from drowsiness, and possibly delicate impairment or depression. The drug is well tolerated and the adverse effects are not shown within the majority of patients. In some patients, though, and in early medical care. Carisoprodol will have a total spectrum of sedative side effects. This can degrade the patient's ability to work on alcoholic drugs, especially once in the form of soft-type small hands, motorcycles and various types of mixed machinery. During which an alternative medicine will be thought of. As soon as medical care continues, the intensity of the side effects of carisoprodol decreases, as is the case with many different drugs.

Interaction of carisoprodol with opioids, basically all opioids and different centrally-acting analgesics, although notably the semi-synthetic category of codeine-derived subgroups (codeine, ethylmorphine, dihydrocodeine, hydrocodone, oxycodone, nicotine, benzylmorphine) anodyne. Derivatives simultaneously acetyldihydrocodeine, dihydrocodeine, nicodicodeine, and others) which possess a given effect, a small dose of opioid Area allows access. This is generally and particularly helpful wherever injury and cramps may be a part of the case. The effect of synergistic is extra help in various painful things, and also, it is particularly helpful in the aliphatic-type opioid-like intoxicants, levomethadone, ketobemidone, phenadoxone, and others. In alcoholic users, negligence has resulted in the combination of hydrocarbons and overdoses of Carisoprodol.

Medicine that rested Meprobamate and various muscles were usually the subjects of misuse in the Fifty and Sixties. Drug cases were reported at the beginning of 1957 and reportable on several occasions since then.

Carisoprodol, minor tranquilizer, and associated medication such as Tibemate can provide physical dependence with long-term use. When the patients who are medically settled with intensive use of medication require hospitalization, then the drug can be withdrawn.

Due to the possibility of additional serious side effects, this drug is on the list to avoid the old.


Carisoprodol begins with rapid, 30-minute action, with a similar effect lasting about 2–6 hours. It is metabolized within the liver through the hemoprotein P450 enzyme isozyme CYP2C19, an estimated 8 hours half-life emitted by the kidney ally colleague. A substantial proportion of carisoprodol is metabolized to a minor tranquilizer, which may be the best-known drug of abuse and dependence; This may be responsible for the misuse potential of carisoprodol.


Carisoprodol may be an acid organic compound. It is a rheumatic mixture of 2 stereoisomers.

Carisoprodol is synthesized by making 2-methyl-2-propyl propane-1,3-diol, equivalent to one molar of gas, to form chloroformate, that salt is made by reacting with isopropylamine. It reacts with ester or Na cyanate gives carisoprodol.

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